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| PMID | Allele | Disease | Population | Drug Names | SNP | Class | Sentence |
|---|---|---|---|---|---|---|---|
| 17620821 | HUMAN LEUKOCYTE ANTIGEN (HUMAN LEUKOCYTE ANTIGEN) | carbamazepine | NA | allopurinol | NA | negation | |
| NA | |||||||
| 18192896 | HLA-B (HLA-B) | nevirapine | Chinese Han | allopurinol | NA | unclassified | |
| NA | |||||||
| 18385790 | HLA-CLASS I (HLA CLASS II) | stevens-johnson syndrome | Japanese | NA | NA | unclassified | |
| hla class i and ii gene polymorphisms in stevens-johnson syndrome with ocular complications in japanese. | |||||||
| 18637831 | HLA-B*15 | phenytoin | Chinese Han | carbamazepine | NA | unclassified | |
| NA | |||||||
| 18637831 | HLA-B*15 | phenytoin | Chinese Han | carbamazepine | NA | negation | |
| NA | |||||||
| 18855540 | HLA-B*15 | stevens-johnson syndrome | Chinese Han | carbamazepine | NA | unclassified | |
| carbamazepine, hla-b*1502 and risk of stevens-johnson syndrome and toxic epidermal necrolysis: us fda recommendations. | |||||||
| 18855540 | HLA-B*15 | stevens-johnson syndrome | Chinese Han | carbamazepine | NA | unclassified | |
| owing to recent data implicating the hla allele b*1502 as a marker for carbamazepine-induced stevens-johnson syndrome and toxic epidermal necrolysis in han chinese, the fda recommends genotyping all asians for the allele. | |||||||
| 19018717 | HLA-B (HLA-B) | stevens-johnson syndrome | Chinese Han | NA | NA | unclassified | |
| hla-b locus in japanese patients with anti-epileptics and allopurinol-related stevens-johnson syndrome and toxic epidermal necrolysis. | |||||||
| 19262378 | HLA-B*15 | phenytoin | Asian | carbamazepine | NA | unclassified | |
| NA | |||||||
| 19474732 | HUMAN LEUKOCYTE ANTIGEN (HUMAN LEUKOCYTE ANTIGEN) | stevens-johnson syndrome | NA | infliximab | NA | unclassified | |
| purpose of reviewthis study summarizes current research and understanding of the pathogenesis of stevens-johnson syndrome (sjs) and toxic epidermal necrolysis (ten) and provides an update on the treatment of these conditions in children.recent findingsthe association of specific human leukocyte antigen subtypes with sjs and ten occurring in certain racial groups to specific drugs has led to recommendations on pretreatment testing. | |||||||
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