Copyright 2024
| PMID | Allele | Disease | Population | Drug Names | SNP | Class | Sentence |
|---|---|---|---|---|---|---|---|
| 33237511 | HLA-G (HLA-G) | undifferentiated connective tissue diseases | NA | NA | NA | unclassified | |
| endothelial progenitor cell cd34+ and cd133+ concentrations and soluble hla-g concentrations during pregnancy and in cord blood in undifferentiated connective tissue diseases compared to controls. | |||||||
| 33237511 | HLA-G (HLA-G) | undifferentiated connective tissue disease | NA | NA | NA | unclassified | |
| the objective of this study is to evaluate endothelial progenitor cells (epcs) cd34+ cd133- and cd34+ cd133+ and soluble hla-g (shla-g) concentrations among undifferentiated connective tissue disease (uctd) subjects, compared to controls, during pregnancy and in cord blood. | |||||||
| 33294176 | HLA-B*15 | lamotrigine | Asian | valproate | NA | unclassified | |
| NA | |||||||
| 33510372 | HLA-CLASS II (HLA CLASS II) | pneumococcal pneumonia | NA | NA | NA | unclassified | |
| proteolytic cleavage of hla class ii by human neutrophil elastase in pneumococcal pneumonia. | |||||||
| 33523393 | HLA-DRB1*04 | dermatophytoses | NA | NA | NA | positive | |
| nonetheless, a retrospective analysis of the hereditary propensity to dermatophytoses revealed at least several proven genetic relationships such as races, card9 deficiency, hla-dr4 and hla-dr8 type and responsible genes encoding interleukin-22, b-defensin 2 and 4 as well as genetic defects in dectin-1, which increased the prevalence of the disease in families and were involved in the inheritance of the proneness in their members. | |||||||
| 33523393 | HLA-DRB1*08 | dermatophytoses | NA | NA | NA | positive | |
| nonetheless, a retrospective analysis of the hereditary propensity to dermatophytoses revealed at least several proven genetic relationships such as races, card9 deficiency, hla-dr4 and hla-dr8 type and responsible genes encoding interleukin-22, b-defensin 2 and 4 as well as genetic defects in dectin-1, which increased the prevalence of the disease in families and were involved in the inheritance of the proneness in their members. | |||||||
| 33572206 | HLA-DR (HLA-DR) | anoxia | NA | NA | NA | lack_of_evidence | |
| anoxia-reoxygenation decreased hla-dr and cd80 but increased icam-1. | |||||||
| 33636637 | HLA-B*08 | microcephaly | NA | NA | NA | negation | |
| free energy estimation further indicated that the mutation from val to ala at p9 of peptide e-247 (dahakrqtv), which was found exclusively in microcephaly samples, did not reduce hla-b8 affinity. | |||||||
| 33905488 | HLA-B*27 | lordosis | NA | NA | NA | positive | |
| rjd was significantly associated with the spa main diagnosis (highest in pspa), a higher prevalence of hla-b27 positivity, enthesitis, tender and swollen joints, crp, cs-dmards, loss of lumbar lordosis and occiput-wall distance > 0. | |||||||
| 33910426 | HLA-G (HLA-G) | acetate | NA | conjugated | NA | unclassified | |
| NA | |||||||
Copyright 2024