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| PMID | Allele | Disease | Population | Drug Names | SNP | Class | Sentence |
|---|---|---|---|---|---|---|---|
| 30509441 | HLA-B*27 | dysbiosis | NA | NA | NA | unclassified | |
| hla-b*27-induced changes in gut microbiota are complex and suggest an ecological model of dysbiosis in rodents. | |||||||
| 31039390 | HLA-B*27 | dysbiosis | NA | NA | NA | unclassified | |
| this article reviews the recent studies on changes of gut microbiota in as patients, and discusses the possible correlation between intestinal dysbiosis and as development from aspects including genetic factor hla-b27, mucosal immune responses and the depression accompanying as. | |||||||
| 31321608 | HLA (HLA) | dysbiosis | NA | NA | NA | unclassified | |
| recent studies have shown that hla haplotype influences the microbiome composition in infants and that dysbiosis in the intestinal microflora, in turn, contributes to loss of tolerance to gluten. | |||||||
| 33973546 | HLA-B*27 | dysbiosis | NA | NA | NA | unclassified | |
| likewise, healthy carriers of human leukocyte antigen (hla)-b27 exhibited gut dysbiosis, indicating that this predisposing allele could exert its pathogenic effect by influencing microbiota composition, and possibly by driving antigen-specific cross-reactive immune response. | |||||||
| 33973546 | HUMAN LEUKOCYTE ANTIGEN (HUMAN LEUKOCYTE ANTIGEN) | dysbiosis | NA | NA | NA | unclassified | |
| likewise, healthy carriers of human leukocyte antigen (hla)-b27 exhibited gut dysbiosis, indicating that this predisposing allele could exert its pathogenic effect by influencing microbiota composition, and possibly by driving antigen-specific cross-reactive immune response. | |||||||
| 30382051 | HLA-CLASS I (HLA CLASS II) | lynch syndrome | NA | NA | NA | positive | |
| pd-l1 and hla class i staining increased after heating samples derived from a case of analinvasion of rectalcancer associated with lynch syndrome. | |||||||
| 27151133 | HLA (HLA) | homologous transplantation | NA | NA | NA | unclassified | |
| homologous transplantation with fresh frozen bone for dental implant placement can induce hla sensitization: a preliminary study. | |||||||
| 27211051 | DQA1*05:01-DQB1*02:01 (HAPLOTYPE) | autoimmune polyendocrine syndrome type 2 | NA | vitamin | NA | lack_of_evidence | |
| among the genetic factors for isolated aad and autoimmune polyendocrine syndrome type 2, a key role is played by hla class ii genes: hla-drb1*0301-dqa1*0501-dqb1*0201 and drb1*04-dqa1*0301-dqb1*0302 are positively, and drb1*0403 is negatively, associated with genetic risk for aad. | |||||||
| 27211051 | HLA-DQA1*03 | autoimmune polyendocrine syndrome type 2 | NA | vitamin | NA | lack_of_evidence | |
| among the genetic factors for isolated aad and autoimmune polyendocrine syndrome type 2, a key role is played by hla class ii genes: hla-drb1*0301-dqa1*0501-dqb1*0201 and drb1*04-dqa1*0301-dqb1*0302 are positively, and drb1*0403 is negatively, associated with genetic risk for aad. | |||||||
| 27211051 | HLA-DRB1*04 | autoimmune polyendocrine syndrome type 2 | NA | vitamin | NA | lack_of_evidence | |
| among the genetic factors for isolated aad and autoimmune polyendocrine syndrome type 2, a key role is played by hla class ii genes: hla-drb1*0301-dqa1*0501-dqb1*0201 and drb1*04-dqa1*0301-dqb1*0302 are positively, and drb1*0403 is negatively, associated with genetic risk for aad. | |||||||
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