Copyright 2024
| PMID | Allele | Disease | Population | Drug Names | SNP | Class | Sentence |
|---|---|---|---|---|---|---|---|
| 28659473 | HLA (HLA) | coinfection | NA | NA | NA | only_studied | |
| proviral dna was longitudinally evaluated in 66 individuals using geno2pheno[coreceptor] demographics, viral load, cd4+ and cd8+ t cell counts, ccr5d32 polymorphisms, gb virus c (gbv-c) coinfection, and hla profiles were also evaluated. | |||||||
| 30944340 | HLA-DR (HLA-DR) | coinfection | Caucasian | NA | NA | unclassified | |
| our objective was to evaluate the influence of hcv coinfection in hiv-1 viral reservoir size in resting (r) cd4+ t-cells (cd25-cd69-hladr-). | |||||||
| 32635221 | HLA-DR (HLA-DR) | coinfection | NA | hepatitis | NA | unclassified | |
| the impact of hcv coinfection on hiv provirus transcription was analyzed in resting (r)cd4 t+ cells (cd3+cd4+cd25-cd69-hladr-) and rcd4 t cells-depleted pbmcs (rcd4 t- pbmcs) from a multicenter cross-sectional study of 115 cart-treated hiv patients: 42 hiv+/hcv+ coinfected individuals (hiv+/hcv+), 34 hiv+ patients with hcv spontaneous clearance (hiv+/hcv-) and 39 hiv patients (hiv+). | |||||||
| 33970847 | HLA-DR (HLA-DR) | coinfection | NA | NA | NA | positive | |
| the frequencies of cd4+ cd38+ hla-dr+ and cd8+ cd38+ hla-dr+ in the hiv/hbv coinfection were significantly higher than hbv monoinfected group (p< 0.0001) and in the hiv monoinfected group (p < 0.0001). | |||||||
| 20191046 | HLA-DQB1 (HLA-DQB1) | myoclonus epilepsy | Korean | NA | NA | unclassified | |
| in a search of clinically plausible causes, brain mri in all patients, mitochondrial dna studies for mitochondrial encephalomyopathy, lactic acidosis, and strokelike episodes (melas) and myoclonus epilepsy and ragged red fibers (merrf) in four patients, and genomic typing on hla drb/hla dqb genes in three patients were performed. | |||||||
| 20191046 | HLA-DQB1 (HLA-DQB1) | merrf | Korean | NA | NA | unclassified | |
| in a search of clinically plausible causes, brain mri in all patients, mitochondrial dna studies for mitochondrial encephalomyopathy, lactic acidosis, and strokelike episodes (melas) and myoclonus epilepsy and ragged red fibers (merrf) in four patients, and genomic typing on hla drb/hla dqb genes in three patients were performed. | |||||||
| 20191046 | HLA-DQB1 (HLA-DQB1) | mitochondrial encephalomyopathy lactic acidosis and strokelike episodes | Korean | NA | NA | unclassified | |
| in a search of clinically plausible causes, brain mri in all patients, mitochondrial dna studies for mitochondrial encephalomyopathy, lactic acidosis, and strokelike episodes (melas) and myoclonus epilepsy and ragged red fibers (merrf) in four patients, and genomic typing on hla drb/hla dqb genes in three patients were performed. | |||||||
| 20191046 | HLA-DRB1*14 | merrf | Korean | NA | NA | unclassified | |
| mitochodrial dna studies for melas and merrf were negative in those children and hla-drb1*1401, hla-drb3*0202, and hla-dqb1*0502 seemed to be significant. | |||||||
| 20191046 | HLA-DRB1*14 | melas | Korean | NA | NA | unclassified | |
| mitochodrial dna studies for melas and merrf were negative in those children and hla-drb1*1401, hla-drb3*0202, and hla-dqb1*0502 seemed to be significant. | |||||||
| 20191046 | HLA-DQB1*05 | merrf | Korean | NA | NA | unclassified | |
| mitochodrial dna studies for melas and merrf were negative in those children and hla-drb1*1401, hla-drb3*0202, and hla-dqb1*0502 seemed to be significant. | |||||||
Copyright 2024