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| PMID | Allele | Disease | Population | Drug Names | SNP | Class | Sentence |
|---|---|---|---|---|---|---|---|
| 26779161 | HLA (HLA) | anthrax | NA | NA | NA | unclassified | |
| these responses were influenced by the specific hla alleles presenting the peptide, and imply that construction of future epitope string vaccines which are immunogenic across a wide range of hla alleles could benefit from a combination of both cryptic and immunodominant anthrax epitopes. | |||||||
| 16872993 | HUMAN LEUKOCYTE ANTIGEN (HUMAN LEUKOCYTE ANTIGEN) | chromosomal disorders | NA | NA | NA | unclassified | |
| it discusses the growing list of indications for pgd including chromosomal disorders, monogenic disorders and human leukocyte antigen typing typing of embryos. | |||||||
| 18158966 | HLA-A (HLA-A) | wilms tumor 1 | NA | NA | NA | unclassified | |
| cd8(+) t cells specific for hla-a * 0201-associated cytomegalovirus (cmv), epstein-barr virus (ebv), and wilms tumor 1 peptide epitopes were retained at significant numbers within the allodepleted donor lymphocyte subsets. | |||||||
| 27181332 | HLA-A*24 | wilms tumor 1 | NA | NA | NA | unclassified | |
| moreover, in the presence of hla-a*24:02-restricted wilms tumor 1 (wt1)235-243 peptide, dcs conditioned by b3a2-specific th cells efficiently stimulated the primary expansion of wti-specific cytotoxic t lymphocytes (ctls). | |||||||
| 27548033 | HLA-A*24 | wilms tumor 1 | NA | NA | NA | unclassified | |
| furthermore, only k562/hla+cd80/86+pd-l1 cells pulsed with hla-a*24:02-restricted wilms tumor 1 (wt1) peptide clearly expanded wt1-specific ctls from healthy donors. | |||||||
| 15823128 | HUMAN LEUKOCYTE ANTIGEN (HUMAN LEUKOCYTE ANTIGEN) | immune system disorders | NA | hepatitis | NA | only_studied | |
| congenital and acquired immune system disorders, viral oncogenes, and various human leukocyte antigen (hla) types were investigated. | |||||||
| 15885065 | HLA-DR (HLA-DR) | gangliogliomas | NA | NA | NA | only_studied | |
| in the present study, the distribution of cells of the microglia/macrophage lineage was studied by immunocytochemistry for cd68 and human leucocyte antigen (hla)-dr in a group of glioneuronal tumours, including gangliogliomas (gg, n = 30), and dysembryoplastic neuroepithelial tumours (dnt, n = 17), from patients with chronic intractable epilepsy. | |||||||
| 15885065 | HLA-DR (HLA-DR) | intractable epilepsy | NA | NA | NA | only_studied | |
| in the present study, the distribution of cells of the microglia/macrophage lineage was studied by immunocytochemistry for cd68 and human leucocyte antigen (hla)-dr in a group of glioneuronal tumours, including gangliogliomas (gg, n = 30), and dysembryoplastic neuroepithelial tumours (dnt, n = 17), from patients with chronic intractable epilepsy. | |||||||
| 15924672 | HLA-G (HLA-G) | fetal growth restriction | NA | NA | NA | only_studied | |
| to investigate the expressions of human histocompatibility antigen-g (hla-g) mrna in placenta of idiopathic fetal growth restriction (ifgr) and its relationship with pathogenesis of ifgr. | |||||||
| 16911023 | NA (NA) | fetal growth restriction | NA | NA | NA | unclassified | |
| the precise nature of these interactions merits further investigation, as knowledge of fetal hla-dq type may be useful in refining risk estimates of severe fetal growth restriction because of maternal smoking during pregnancy. | |||||||
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